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PERAMPANEL-BTRE-META

Efficacy and Safety of Perampanel in Patients With Brain Tumor-Related Epilepsy: A Systematic Review and Meta-Analysis

Year of Publication: 2026

Authors: Da Silva AMP, Gonçalves OR, Tudella GCN, et al.

Journal: Neurology Clinical Practice

Citation: Neurol Clin Pract 2026;16(4):e200636. DOI: 10.1212/CPJ.0000000000200636

Link: https://doi.org/10.1212/CPJ.0000000000200636

Bottom Line

Across 8 observational studies (n=382), perampanel achieved seizure freedom in 42% and ≥50% seizure reduction in 46% of BTRE patients, with adverse events in 26% (mostly mild-to-moderate) and no signal of an effect on tumor progression — supporting perampanel as a mechanistically targeted, non–enzyme-inducing option for BTRE.

Major Points

  • Systematic review and meta-analysis of 8 observational studies (published 2017–2024) including 382 adults with brain tumor-related epilepsy treated with perampanel
  • Pooled seizure freedom rate 42% (95% CI 27–59%; I²=65.7%, 7 studies); ≥50% seizure reduction 46% (95% CI 31–62%; I²=84.3%, 8 studies)
  • Pooled oncologic progression 39% (95% CI 17–65%; I²=0%, 5 studies) — consistent with natural tumor history, no evidence of perampanel effect on tumor behavior
  • Pooled adverse-event rate 26% (95% CI 17–38%; I²=61.6%, 8 studies); most frequent: dizziness, somnolence, fatigue
  • Meta-regression: retrospective design associated with higher seizure-freedom rates (coef 1.03, p=0.035); AEs lower in retrospective cohorts (coef −1.03, p=0.040) and higher in IDH-mutant cases (coef −1.57, p=0.044); sample size and mean age were not associated with any outcome
  • Mechanistic rationale: perampanel is a selective noncompetitive AMPA-receptor antagonist targeting glutamate-mediated peritumoral hyperexcitability that drives BTRE
  • Non–enzyme-inducing profile makes perampanel attractive for co-administration with antineoplastic therapies (minimal drug–drug interactions)
  • Limitations: all included studies were single-arm observational series, small samples, short/variable follow-up, English-only, and heterogeneous outcome definitions — precluding causal inference

Design

Study Type: Systematic Review and Meta-Analysis

Randomization:

Blinding: Not applicable (meta-analysis of single-arm observational studies)

Enrollment Period: Studies published 2017–2024; database search inception to April 2025

Follow-up Duration: Variable across included studies (not standardized)

Centers: 8

Countries: Italy, USA, Austria, Japan, France

Sample Size: 382

Analysis: Random-effects meta-analysis of single proportions (REML estimator); leave-one-out sensitivity, GLMM sensitivity, meta-regression; Egger test and funnel plots for publication bias; PROSPERO CRD420251113758; PRISMA 2020


Inclusion Criteria

  • Prospective or retrospective observational study published in English
  • Adult patients diagnosed with brain tumor-related epilepsy (BTRE) — epileptic seizures in association with a primary or secondary brain tumor
  • Evaluated perampanel as monotherapy or adjunctive antiseizure medication
  • Single-arm data (no control group required)
  • Reported at least one efficacy or safety outcome (seizure freedom, ≥50% seizure reduction, oncologic progression, or adverse events)

Exclusion Criteria

  • Non-original publications (reviews, editorials, commentaries, conference abstracts, theses)
  • Animal or preclinical studies
  • Publications with overlapping populations (retained the most complete/updated dataset)
  • Non-English publications

Baseline Characteristics

CharacteristicOverall (8 studies, N=382)Largest cohort - Nakai 2024 (n=272)PERADET - Coppola 2020 (n=36)
Studies included8 observational (2017–2024)
Total patients382
Sex - Male218 (57.0%)144 (52.9%)23 (63.9%)
Mean age range across studies (y)43–58
Tumor histology - Low-grade glioma (range)17–50%
Tumor histology - High-grade glioma (range)25–80%
Tumor histology - Glioblastoma (range)16.7–27.2%
Tumor location - Frontal (range)25–54.5%
Tumor location - Temporal (range)11.5–28.3%
Tumor location - Parietal (range)8.3–25%
IDH1 mutated (range)16.7–62.5%
IDH1 not mutated (range)25–63.6%
Baseline seizure frequency (mean, per month)9.1–13.6
Perampanel mean dose across studies (mg/day)3.36–7.45
Sample size27236
Tumor location - Frontal41.2%
Tumor location - Temporal28.3%
Tumor location - Parietal19.5%
Focal to bilateral tonic-clonic20.2%
Focal impaired awareness34.2%
Focal aware29.8%
Monotherapy / Polytherapy (%)57.0 / 43.052.8 / 47.2
Perampanel mean dose (mg/day)3.366.53
Mean age (y)45.3 ± 44.5
LGG / HGG / GBM / Other30.6% / 38.9% / 19.4% / 11.1%
IDH1 mutated / not mutated / unknown16.7% / 27.8% / 55.6%
Baseline seizure freq (per month)9.1 ± 12.8

Arms

FieldPerampanel (pooled single-arm)
InterventionPerampanel, monotherapy or adjunctive; mean daily dose ~3.4–7.5 mg/day across studies
DurationVariable across included studies

Outcomes

OutcomeTypeControlInterventionHR / OR / RRP-value
Seizure freedom — proportion of patients seizure-free during follow-up (pooled random-effects meta-analysis; 7 studies)PrimaryN/A (single-arm)42% pooledN/A
≥50% reduction in seizure frequency (8 studies)SecondaryN/A46% (95% CI 31%–62%); I²=84.3%N/A
Oncologic progression during perampanel therapy (5 studies)SecondaryN/A39% (95% CI 17%–65%); I²=0.0% (no signal of perampanel effect on tumor behavior)N/A
Any adverse event (8 studies)SecondaryN/A26% (95% CI 17%–38%); I²=61.6%N/A
Seizure freedom heterogeneity (I²)SecondaryN/A65.7% (substantial); leave-one-out omitting Coppola 2020 resolved heterogeneity to I²=0%N/A
Meta-regression — study design as moderator of seizure freedomSecondaryN/ACoefficient 1.03 (retrospective > prospective)0.0345
Meta-regression — study design as moderator of AEsSecondaryN/ACoefficient −1.03 (lower AE rates in retrospective cohorts)0.0398
Meta-regression — IDH mutation status as moderator of AEsSecondaryN/ACoefficient −1.57 (higher AE probability in IDH-mutant)0.0441
Any adverse event (pooled)Adverse26% (95% CI 17–38%); I²=61.6%
Most common AEsAdverseDizziness, somnolence, fatigue (mild-to-moderate; consistent with known perampanel safety profile)
Serious AEsAdverseNot systematically reported across studies
Discontinuation for AEsAdverseNot pooled
Meta-regression — sample size as moderator of AEsAdverseCoefficient −0.60, p=0.32 (NS)
Meta-regression — mean age as moderator of AEsAdverseCoefficient −0.97, p=0.17 (NS)

Subgroup Analysis

Leave-one-out sensitivity confirmed robustness of all outcomes (seizure freedom 0.37–0.50; ≥50% reduction 0.38–0.49; oncologic progression 0.32–0.44; AEs 0.26–0.30). GLMM sensitivity analyses produced comparable pooled estimates. Meta-regression covariates examined: study design, sample size, mean age, IDH mutation status.


Criticisms

  • All included studies were single-arm observational series — no control groups, precluding causal inference or estimation of relative treatment effects
  • Small individual sample sizes (median cohort ~12; range 5–272) and relatively short follow-up periods
  • Substantial statistical heterogeneity for seizure freedom (I²=65.7%) and ≥50% reduction (I²=84.3%)
  • Heterogeneity in outcome definitions, follow-up duration, and adverse-event reporting across studies (measurement/reporting bias)
  • Inconsistent reporting of perampanel dose, treatment duration, concomitant ASMs, and molecular tumor markers (IDH1, MGMT)
  • Restriction to English-language publications introduced potential language bias
  • Wide confidence intervals for some pooled estimates reflecting limited data
  • Modest number of studies (k<10) limits reliability of publication-bias assessment (funnel plot, Egger test) and statistical power of meta-regression
  • JBI critical appraisal flagged incomplete/non-consecutive inclusion of participants in several retrospective series (selection bias)
  • One study (Nakai 2024) contributed the majority of patients (272/382, 71%) and dominated pooled AE and ≥50%-reduction estimates

Funding

No targeted funding reported. Authors report no relevant disclosures.

Based on: PERAMPANEL-BTRE-META (Neurology Clinical Practice, 2026)

Authors: Da Silva AMP, Gonçalves OR, Tudella GCN, et al.

Citation: Neurol Clin Pract 2026;16(4):e200636. DOI: 10.1212/CPJ.0000000000200636

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