← Back
NeuroTrials.ai
Neurology Clinical Trial Database

DexEnceph

Safety and efficacy of adjunct dexamethasone in adults with herpes simplex virus encephalitis in the UK (DexEnceph): a multicentre, observer-blind, randomised, phase 3, controlled trial

Year of Publication: 2026

Authors: Solomon T, Hooper C, Easton A, ..., Ellul MA

Journal: Lancet Neurology

Citation: Lancet Neurol. 2026;25:136-146


Clinical Question

Does adjunct dexamethasone improve verbal memory outcomes in adults with herpes simplex virus encephalitis treated with aciclovir?

Bottom Line

Adding dexamethasone to standard aciclovir treatment did not improve verbal memory at 26 weeks in adults with HSV encephalitis. The trial was safe with no evidence of harm from dexamethasone and no increase in viral persistence. Routine use of adjunct dexamethasone in HSV encephalitis is not supported; however, a post-hoc signal suggesting earlier treatment may be more effective warrants further study.

Major Points

  • Adjunct dexamethasone (10 mg IV four times daily for 4 days) did not significantly improve verbal memory at 26 weeks compared to aciclovir alone (adjusted difference 1.77 points, 95% CI -9.57 to 13.12, p=0.76).
  • No significant benefit was found in any secondary neuropsychological domain (visual memory, working memory, processing speed, executive function) or clinical outcomes (mortality, seizure rate, time to discharge) at 26 or 78 weeks.
  • Dexamethasone was safe: adverse event rates were similar between groups, there were no treatment-related deaths, and there was no significant increase in HSV PCR positivity in CSF at 2 weeks (11% vs 21%, p=0.25).
  • 5 clinical relapses occurred, all in the dexamethasone group -- potentially explained by early corticosteroid suppression of the immune response to HSV before sufficient viral clearance, with subsequent autoimmune activation.
  • Important post-hoc finding: each day of delay to dexamethasone initiation was associated with a 4.89-point reduction in verbal memory score (95% CI 2.70-7.08, p<0.0001), suggesting that if corticosteroids are beneficial, timing may be critical.
  • The 95% CI for the primary outcome (-9.57 to 13.12) includes the possibility of a clinically meaningful benefit (15.5-point threshold), meaning the trial may have been underpowered to exclude a real benefit.
  • Findings are consistent with the hypothesis that early corticosteroid treatment in all suspected encephalitis (before HSV is excluded by CSF PCR) is unlikely to cause harm, and may support empiric use in appropriate patients.

Design

Study Type: Multicentre, observer-blind, randomised, phase 3, controlled trial

Randomization: 1

Blinding: Observer-blind (neuropsychologist assessing primary outcome and statisticians masked; patients, site teams not masked)

Allocation: 1:1 (minimisation algorithm incorporating age, hospital GCS score, steroid use since admission, delay in aciclovir)

Enrollment Period: September 9, 2016 – February 21, 2022

Follow-up Duration: 78 weeks (primary outcome at 26 weeks)

Centers: 53

Countries: UK

Sample Size: 94

Analyzed: 81

Analysis: Modified intention-to-treat; linear regression for primary outcome adjusted for minimisation factors

Power Calculation: Sample of 45 per group (90 total) to detect 15.5-point difference in WMS-IV verbal memory with 80% power at alpha=0.05, assuming 10% mortality before 26-week assessment

Registration: ISRCTN11774734; EudraCT2016-004835-19


Inclusion Criteria

  • Age 16 years or older
  • Suspected diagnosis of encephalitis (new-onset seizure or new focal neurological signs or altered consciousness, cognition, personality, or behaviour)
  • HSV type 1 or 2 DNA positive in CSF by PCR, reported within 7 days before randomisation
  • Receiving intravenous aciclovir 10 mg/kg three times daily

Exclusion Criteria

  • Oral or injectable corticosteroid therapy within the 30 days before study entry (except topical or inhaled)
  • History of hypersensitivity to corticosteroids
  • Immunosuppression (HIV with CD4 <200/mm3, biological therapy, immunosuppressive agents, solid organ transplant with ongoing immunosuppression, bone marrow transplant, current chemotherapy/radiotherapy, primary immunodeficiency, haematological malignancy)
  • Pre-existing indwelling ventricular devices
  • Active peptic ulcer disease within past 6 months
  • Current antiretroviral regimen containing rilpivirine without feasible switch

Arms

FieldDexamethasone + AciclovirControl
N4747
InterventionIV dexamethasone 10 mg four times daily for 4 days (starting within 24 hours of randomisation) plus IV aciclovir 10 mg/kg three times daily for at least 14 daysIV aciclovir 10 mg/kg three times daily for at least 14 days (standard care)
Duration4 days dexamethasone; aciclovir continued until HSV PCR negative in CSFAt least 14 days (continued until HSV PCR negative)

Outcomes

OutcomeTypeControlInterventionHR / OR / RRP-value
Verbal memory score at 26 weeks, measured by the Wechsler Memory Scale (WMS-IV) Auditory Memory IndexPrimary69 (SD 25) [n=42]71 (SD 26) [n=39]0.76
Visual memory (WMS-IV) at 26 weeksSecondary71 (25) [n=31]69 (25) [n=33]0.86
Immediate memory (WMS-IV) at 26 weeksSecondary67 (24) [n=31]71 (25) [n=33]0.85
Delayed memory (WMS-IV) at 26 weeksSecondary65 (24) [n=31]68 (24) [n=33]1.0
Working memory (WAIS-IV) at 26 weeksSecondary82 (27) [n=36]85 (25) [n=36]0.47
Processing speed (WAIS-IV) at 26 weeksSecondary77 (26) [n=32]80 (23) [n=33]0.50
All-cause mortality at 78 weeksSecondary6/47 (13%)6/47 (13%)
Seizure frequency at 78 weeksSecondary10/38 (26%)7/36 (19%)0.55
Time to hospital discharge (weeks)SecondaryMedian 4 (IQR 2-6) [n=45]Median 3 (IQR 2-8) [n=47]0.75
HSV DNA detectable in CSF at 2 weeks (secondary safety)Secondary9/43 (21%)4/36 (11%)0.25
Clinical relapseSecondary05 (all in dexamethasone group)
Notable adverse eventsSafety18/47 (40%)18/47 (38%)
Serious adverse eventsSafety5/47 (11%)7/47 (15%)
Treatment-related deathsSafety00
Neuropsychiatric disordersAdverse5 (11%)7 (15%)
Seizure (SAE)Adverse1 (2%)1 (2%)
Autoimmune encephalitis or meningoencephalitisAdverse03 (6%)
Deep vein thrombosisAdverse01 (2%)
Pulmonary embolismAdverse01 (2%)
Acute kidney injuryAdverse01 (2%)

Subgroup Analysis

No prespecified subgroup analysis demonstrated a benefit of dexamethasone. A post-hoc analysis found that earlier initiation of dexamethasone was associated with better verbal memory outcomes: each additional day of delay was associated with a 4.89-point reduction in WMS-IV score (95% CI 2.70-7.08, p<0.0001). This association was significant in the dexamethasone group but not in the control group, suggesting a time-sensitive treatment window. This is exploratory and hypothesis-generating.


Criticisms

  • Small sample size (n=81 in modified ITT) -- underpowered for secondary outcomes; primary outcome 95% CI includes potential clinically meaningful benefit
  • Recruitment took 5.5 years across 53 hospitals, reflecting the rarity of HSV encephalitis and enrollment challenges
  • Observer-blind rather than double-blind -- patients and site teams were aware of allocation, introducing potential performance and detection bias
  • Delayed enrollment: median 7-8 days from hospital admission to randomisation -- corticosteroids may be most effective in early inflammation; this could have masked benefit
  • 5 clinical relapses all occurred in the dexamethasone group, raising concern about post-viral autoimmune phenomena potentially triggered by early immune suppression
  • COVID-19 pandemic required remote assessments for some participants, reducing neuropsychological data quality
  • All participants were recruited in the UK -- predominantly White population (>90%), limiting generalisability to diverse populations

Funding

National Institute for Health and Care Research (NIHR) Efficacy and Mechanism Evaluation Programme (reference 12/205/28); NIHR Global Health Research Group on Brain Infections (17/63/110)

Based on: DexEnceph (Lancet Neurology, 2026)

Authors: Solomon T, Hooper C, Easton A, ..., Ellul MA

Citation: Lancet Neurol. 2026;25:136-146

Content summarized and formatted by NeuroTrials.ai.