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Neurology Clinical Trial Database

RE-SPECT CVT

Safety and Efficacy of Dabigatran Etexilate vs Dose-Adjusted Warfarin in Patients With Cerebral Venous Thrombosis

Year of Publication: 2019

Authors: José M. Ferro, MD, PhD; Jonathan M. Coutinho, ..., PhD; Hans-Christoph Diener

Journal: JAMA Neurology

Citation: JAMA Neurol. 2019;76(12):1457-1465.

Link: https://doi.org/10.1001/jamaneurol.2019.2764


Clinical Question

Is dabigatran etexilate as efficacious and safe as dose-adjusted warfarin for preventing recurrent venous thrombotic events (VTEs) in patients with a recent cerebral venous thrombosis (CVT)?

Bottom Line

In this exploratory randomized trial, patients with CVT treated with either dabigatran or warfarin had a very low risk of recurrent VTE, with zero events observed in either group. The risk of major bleeding was also low and similar between the two medications, suggesting dabigatran may be a safe and effective alternative to warfarin for preventing VTE recurrence after CVT, although the study was not powered for noninferiority.

Major Points

  • First and only randomized trial comparing a DOAC to warfarin for cerebral venous thrombosis: 120 patients across 51 centers in 9 countries — an exploratory, hypothesis-generating study.
  • Zero recurrent VTEs in EITHER group during 24 weeks — confirming that both dabigatran and warfarin effectively prevent CVT recurrence in the acute/subacute phase.
  • Major bleeding was infrequent and similar: 1.7% dabigatran vs 3.3% warfarin — suggesting comparable safety profiles for CVT anticoagulation.
  • Cerebral venous recanalization rates were similar (60.0% dabigatran vs 67.3% warfarin) — no evidence that warfarin's mechanism provides superior venous recanalization.
  • Higher treatment discontinuation with dabigatran (11.7% vs 0% with warfarin) was unexpected — primarily due to GI side effects, raising adherence concerns specific to CVT patients.
  • Not powered for noninferiority — the trial was explicitly exploratory, so equivalence between dabigatran and warfarin cannot be formally concluded.
  • Mild CVT population (80% had NIHSS 0; only 30% had hemorrhagic lesions) — results may not apply to severe CVT with large parenchymal hemorrhage or coma.
  • First RCT evidence supporting DOAC use in CVT, filling a critical evidence gap — most CVT guidelines previously had no randomized data to inform DOAC recommendations.
  • Changed clinical practice: many CVT experts now use DOACs based on RE-SPECT CVT despite its limitations, particularly for patients with mild CVT and no parenchymal hemorrhage.
  • Led to EAN/ESO guidelines acknowledging DOACs as a reasonable alternative to warfarin in CVT — with the caveat that evidence is limited to this single exploratory trial.

Design

Study Type: Exploratory, prospective, randomized (1:1), parallel-group, open-label, multicenter clinical trial with blinded end-point adjudication (PROBE design).

Randomization: 1

Blinding: Open-label with blinded endpoint adjudication.

Enrollment Period: December 21, 2016, to June 22, 2018.

Follow-up Duration: 24 weeks.

Centers: 51

Countries: France, Germany, India, Italy, the Netherlands, Poland, Portugal, Russia, Spain

Sample Size: 120

Analysis: Intention-to-treat.


Inclusion Criteria

  • Adult consecutive patients (18-79 years) with acute CVT confirmed by imaging.
  • Clinically stable after 5 to 15 days of initial treatment with parenteral heparin.

Exclusion Criteria

  • CVT associated with central nervous system infection or major head trauma.
  • Planned surgical procedure for CVT (e.g., decompressive hemicraniectomy).
  • Creatinine clearance level less than 30 mL/min.
  • Life-threatening or major bleeding in the previous 6 months (other than intracranial hemorrhage from the index CVT).

Arms

FieldControlDabigatran
InterventionDose-adjusted warfarin to maintain an international normalized ratio (INR) between 2.0 and 3.0. Dabigatran 150 mg twice daily.
Duration24 weeks 24 weeks

Outcomes

OutcomeTypeControlInterventionHR / OR / RRP-value
A composite of a new venous thrombotic event (VTE) or a major bleeding event during the 24-week study period. Primary3.3% (2 events) 1.7% (1 event) 1.60%No formal hypothesis testing performed.
All venous thrombotic eventsSecondary0% 0%
Cerebral venous recanalization (improved score)Secondary67.3% (35/52) 60.0% (33/55)
Major bleeding eventAdverse3.3% (2/60) 1.7% (1/60)
Any bleeding eventAdverse20.0% (12/60) 20.0% (12/60)
Adverse event leading to trial drug discontinuationAdverse0% (0/60) 11.7% (7/60)

Criticisms

  • Exploratory trial with only 120 patients — severely underpowered to establish noninferiority or detect clinically meaningful differences between dabigatran and warfarin.
  • Open-label design may bias management decisions (e.g., threshold for ordering imaging, managing complications) despite blinded endpoint adjudication.
  • Predominantly mild CVT population (80% NIHSS 0, only 30% with hemorrhagic lesions) — results cannot be extrapolated to severe CVT with coma, large hemorrhage, or mass effect.
  • Zero recurrent VTEs in both groups means the study had no statistical power to compare efficacy — the event rate was far lower than anticipated.
  • Higher dabigatran discontinuation (11.7% vs 0%) raises adherence concerns — in real-world practice, this dropout rate could translate to worse outcomes.
  • 24-week duration is short — many CVT patients require 3–12+ months of anticoagulation; long-term DOAC safety in CVT is unknown.
  • Industry-sponsored by Boehringer Ingelheim (dabigatran manufacturer) — potential conflict of interest in an exploratory trial that could influence prescribing.
  • Excluded severe CVT cases (those needing decompressive surgery or with active CNS infection) — the highest-risk patients where anticoagulation decisions are most critical were not studied.
  • No guidance on dose adjustment for renal function or drug interactions — dabigatran pharmacokinetics may differ in CVT patients with cerebral edema and altered drug metabolism.

Funding

Boehringer Ingelheim.

Based on: RE-SPECT CVT (JAMA Neurology, 2019)

Authors: José M. Ferro, MD, PhD; Jonathan M. Coutinho, ..., PhD; Hans-Christoph Diener

Citation: JAMA Neurol. 2019;76(12):1457-1465.

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