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TenCRAOS

Tenecteplase in Central Retinal Artery Occlusion Study

Year of Publication: 2026

Authors: S.J. Ryan, Ø.K. Jørstad, M. Skjelland, ..., for the TenCRAOS Investigators

Journal: New England Journal of Medicine

Citation: N Engl J Med 2026;394:442-50

Link: https://doi.org/10.1056/NEJMoa2508515


Clinical Question

Does intravenous tenecteplase administered within 4.5 hours of symptom onset improve visual outcomes compared to oral aspirin in patients with acute central retinal artery occlusion?

Bottom Line

Intravenous tenecteplase at 0.25 mg/kg administered within 4.5 hours of symptom onset did not result in significantly greater vision recovery than oral aspirin 300 mg in patients with central retinal artery occlusion, and was associated with increased serious adverse events including one fatal intracranial hemorrhage. Routine thrombolytic therapy for central retinal artery occlusion is not supported.

Major Points

  • Phase 3, double-blind, double-dummy RCT comparing IV tenecteplase 0.25 mg/kg to oral aspirin 300 mg in acute central retinal artery occlusion within 4.5 hours of symptom onset
  • Primary endpoint (BCVA ≤0.7 logMAR at 30 days) was achieved by 20% in tenecteplase group vs 24% in aspirin group (risk difference -3.7 percentage points; 95% CI -22.0 to 14.7; P=0.69)
  • No substantial differences in secondary visual endpoints including BCVA ≤0.5 logMAR (20% vs 18%) and mean BCVA improvement
  • Subgroup analysis of patients treated within 3 hours (69% of population) showed no benefit: 23% vs 22% achieved primary endpoint
  • One fatal intracranial hemorrhage occurred in the tenecteplase group due to hemorrhagic transformation of unrecognized cerebral ischemia
  • More adverse events (30 vs 19) and serious adverse events (10 vs 4) in tenecteplase group
  • Results consistent with prior THEIA trial of IV alteplase and EAGLE trial of intraarterial thrombolysis
  • Central retinal artery occlusion appears resistant to reperfusion strategies effective in cerebral ischemia despite similar pathophysiology

Design

Study Type: Phase 3, double-blind, double-dummy, parallel-group, randomized controlled trial

Randomization: 1

Blinding: Double-blind with double-dummy design; patients, investigators, and outcome assessors were blinded to treatment allocation

Enrollment Period: November 8, 2020 to March 16, 2025

Follow-up Duration: 30 days (±5 days)

Centers: 16

Countries: Norway, Denmark, Finland, Belgium, Lithuania, Sweden

Sample Size: 78

Analysis: Logistic regression with treatment group as covariate; primary analysis in full analysis population (received assigned treatment within 4.5 hours); population-averaged marginal risk difference with 95% CI; sensitivity analyses including intention-to-treat, complete case, worst-case and best-case imputation; R software version 4.5.0


Inclusion Criteria

  • Adults ≥18 years of age
  • Diagnosis of central retinal artery occlusion based on typical history and acute ophthalmologic examination
  • Best corrected visual acuity (BCVA) ≥1.0 logMAR in the affected eye (equivalent to decimal BCVA ≤0.1 or fraction BCVA ≤20/200)
  • Able to receive trial treatment within 4.5 hours after symptom onset
  • Assessed by ophthalmologist and acute stroke team
  • Written informed consent obtained

Exclusion Criteria

  • Arteritic central retinal artery occlusion (assessed clinically due to time constraints)
  • Concomitant intracranial vascular event on neuroimaging
  • Cilioretinal artery supplying the macula preserving BCVA
  • Retinal vasospasm rather than central retinal artery occlusion
  • Standard contraindications to thrombolysis (implied by stroke protocol adherence)

Arms

FieldTenecteplaseControl
InterventionIntravenous tenecteplase at a dose of 0.25 mg per kilogram of body weight plus oral placebo capsule (dummy comedication)Intravenous placebo (dummy treatment) plus oral aspirin capsule at a dose of 300 mg
DurationSingle administration within 4.5 hours of symptom onsetSingle administration within 4.5 hours of symptom onset

Outcomes

OutcomeTypeControlInterventionHR / OR / RRP-value
BCVA of 0.7 logMAR or lower in the affected eye at 30 days after treatment (equivalent to decimal BCVA ≥0.2 or fraction BCVA ≥20/100), reflecting an improvement of at least 0.3 logMAR (15 letters on ETDRS chart)Primary9/38 (24%)8/40 (20%)0.69
BCVA ≤0.5 logMAR at 30 days (equivalent to ≥20/63)Secondary7/38 (18%)8/40 (20%)
Mean change in BCVA from baseline to 30 days (logMAR)Secondary-0.60 ± 0.91-0.73 ± 0.92
Mean Esterman perimetry score at 30 days (test points out of 100)Secondary37 ± 3835 ± 36
Mean NIHSS score at dischargeSecondary0 ± 0.20.1 ± 0.3
Mean NIHSS score at 30 daysSecondary00.2 ± 0.5
Mean mRs score at dischargeSecondary1.3 ± 0.81.3 ± 1.1
Mean mRs score at 30 daysSecondary1.4 ± 1.01.4 ± 0.3
Mean EQ-5D-5L index value at 30 daysSecondary0.87 ± 0.130.84 ± 0.25
Mean VFQ-25 score at 30 daysSecondary74.1 ± 18.674.6 ± 20.3
Neovascularization in affected eye at 30 daysSecondary1/38 (3%)1/40 (2%)
BCVA ≤0.7 logMAR in subgroup treated ≤3 hoursSecondary5/23 (22%)7/31 (23%)
BCVA ≤0.5 logMAR in subgroup treated ≤3 hoursSecondary4/23 (17%)7/31 (23%)
Improvement in BCVA of ≥0.3 logMAR (including off-chart improvements)Secondary19/38 (50%)25/40 (62%)
Death at 30 daysAdverse0 (0%)1 (2%)
Symptomatic intracranial hemorrhageAdverse0 (0%)1 (2%)
Any intracranial hemorrhage at 24 hoursAdverse0 (0%)2 (5%)
Any systemic bleeding at 30 daysAdverse2 (5%)1 (2%)
Ischemic strokeAdverse1 (3%)1 (2%)
Reocclusion of central retinal arteryAdverse0 (0%)1 (2%)
Giant-cell arteritisAdverse0 (0%)1 (2%)
Carotid endarterectomy or stentingAdverse2 (5%)3 (8%)
Hypotension or syncopeAdverse1 (3%)1 (2%)
Any adverse event (patients)Adverse13 (34%)19 (48%)
Serious adverse events (events in patients)Adverse4 events in 4 patients10 events in 8 patients

Subgroup Analysis

Subgroup analysis by time from symptom onset to treatment (≤3 hours vs 3-4.5 hours) showed no benefit in either subgroup. In patients treated within 3 hours (69% of population), 23% in tenecteplase group vs 22% in aspirin group achieved BCVA ≤0.7 logMAR (risk difference 0.8 percentage points; 95% CI -21.5 to 23.2). Subgroup analysis by cause of central retinal artery occlusion was also performed but detailed results not reported in main text.


Criticisms

  • Small sample size (78 patients) with statistical power to detect only a 30 percentage-point difference; trial was underpowered to detect smaller but potentially clinically meaningful differences
  • Trial was powered based on 2015 meta-analytic data suggesting large treatment effect, but later observational data showed no superiority of thrombolysis
  • Mean change in BCVA required conversion of no light perception and light perception to logMAR values, which is inherently inaccurate
  • Time of symptom onset relied solely on patient report
  • 95% confidence interval for primary endpoint includes up to 14.7 percentage points in favor of tenecteplase, so modest benefit cannot be ruled out
  • Single fatal event should be interpreted in broader context of bleeding risk in stroke and prior thrombolysis trials
  • Baseline imbalances in carotid artery disease (higher in aspirin group) and diabetes mellitus (higher in tenecteplase group)
  • Unable to rule out arteritic central retinal artery occlusion with laboratory/imaging within 4.5-hour window; relied on clinical assessment

Funding

Oslo University Hospital, Norwegian Program for Clinical Therapy Research in the specialist health service (Klinbeforsk), South-Eastern Norway Regional Health Authority, International Order of Odd Fellows, and unconditional grant from Boehringer Ingelheim Norway for purchase of tenecteplase

Based on: TenCRAOS (New England Journal of Medicine, 2026)

Authors: S.J. Ryan, Ø.K. Jørstad, M. Skjelland, ..., for the TenCRAOS Investigators

Citation: N Engl J Med 2026;394:442-50

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