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Verubecestat-Prodromal

Randomized Trial of Verubecestat for Prodromal Alzheimer's Disease

Year of Publication: 2019

Authors: Michael F. Egan, James Kost, Tiffini Voss, ..., David Michelson

Journal: New England Journal of Medicine

Citation: N Engl J Med 2019;380:1408-20

Link: https://doi.org/10.1056/NEJMoa1812840


Clinical Question

Can verubecestat, a BACE-1 inhibitor that reduces amyloid-beta production, slow disease progression in patients with prodromal Alzheimer's disease (MCI with elevated brain amyloid)?

Bottom Line

Verubecestat did not improve clinical ratings of dementia among patients with prodromal Alzheimer's disease, and some measures suggested that cognition and daily function were worse among patients who received verubecestat than among those who received placebo, despite reducing brain amyloid levels

Major Points

  • Phase 3 trial terminated early for futility after interim analysis
  • Primary outcome not met: CDR-SB worsening was similar or greater with verubecestat vs placebo
  • 40mg dose showed statistically worse outcome than placebo on CDR-SB (2.02 vs 1.58, p=0.01)
  • Higher progression rate to dementia with verubecestat (HR 1.38 for 40mg vs placebo)
  • Verubecestat effectively reduced brain amyloid on PET (decreased 0.04 SUVR vs increased 0.02 with placebo)
  • Cerebrospinal fluid Aβ42 reduced >60% with verubecestat confirming target engagement
  • Multiple secondary outcomes suggested worse cognition/function with verubecestat
  • Cognitive worsening apparent by week 13 and did not progress further
  • Adverse events more common with verubecestat including rash, sleep disturbance, weight loss, hair color change

Design

Study Type: Randomized, double-blind, placebo-controlled, parallel group phase 3 trial

Randomization: 1

Blinding: Double-blind; participants, investigators, and assessors blinded; audiorecorded assessments with central rater quality review

Enrollment Period: November 2013 to April 2018

Follow-up Duration: 104 weeks (planned); terminated early for futility

Centers: 238

Countries: United States, Canada, Europe (multiple), Australia, New Zealand, Japan

Sample Size: 1454

Analysis: Longitudinal ANCOVA with Bonferroni correction; hierarchical sequential testing; modified intention-to-treat


Inclusion Criteria

  • Age 50-85 years
  • Subjective decrease in memory for at least 1 year corroborated by informant
  • RBANS Delayed Memory Index score ≤85 (≥1 SD below age/education-appropriate mean)
  • Positive brain amyloid on visual inspection of amyloid-ligand PET
  • MMSE score 24-30
  • Did not meet criteria for dementia
  • Diagnosis of prodromal Alzheimer's disease confirmed by independent review
  • Stable dose of AChEI or memantine for ≥3 months if taking

Exclusion Criteria

  • Meets criteria for dementia
  • Other causes of cognitive impairment

Arms

FieldVerubecestat 12mgVerubecestat 40mgControl
InterventionVerubecestat 12mg orally once dailyVerubecestat 40mg orally once dailyMatching placebo orally once daily
Duration104 weeks (planned); terminated early104 weeks (planned); terminated early104 weeks (planned); terminated early

Outcomes

OutcomeTypeControlInterventionHR / OR / RRP-value
Change in CDR-SB score from baseline to week 104 (range 0-18, higher scores indicate worse cognition and daily function)Primary1.5812mg vs placebo: 0.67; 40mg vs placebo: 0.01 (favoring placebo)
Progression to dementia due to Alzheimer's disease (events per 100 patient-years)Secondary19.312mg: 24.5; 40mg: 25.512mg: HR 1.30 (97.51% CI 1.01-1.68); 40mg: HR 1.38 (97.51% CI 1.07-1.79)
CCS-3D change at 104 weeksSecondary0.7712mg: 0.77; 40mg: 0.78
Hippocampal volume change at 104 weeks (%)Secondary-6.1%12mg: -6.5%; 40mg: -6.7%
Cortical amyloid SUVR change at 104 weeksSecondary+0.0212mg: -0.03; 40mg: -0.04
ADCS-ADLMCI change at 104 weeksSecondary-4.112mg: -5.2; 40mg: -5.8
Any adverse eventAdverse421 (87.0%)12mg: 441 (91.3%); 40mg: 446 (92.1%)
Serious adverse eventAdverse96 (19.8%)12mg: 124 (25.7%); 40mg: 101 (20.9%)
DeathAdverse3 (0.6%)12mg: 3 (0.6%); 40mg: 1 (0.2%)
Rash, dermatitis, urticariaAdverse62 (12.8%)12mg: 96 (19.9%); 40mg: 101 (20.9%)
DepressionAdverse25 (5.2%)12mg: 32 (6.6%); 40mg: 50 (10.3%)
AnxietyAdverse21 (4.3%)12mg: 33 (6.8%); 40mg: 44 (9.1%)
Sleep disturbanceAdverse22 (4.5%)12mg: 38 (7.9%); 40mg: 44 (9.1%)
Weight lossAdverse10 (2.1%)12mg: 27 (5.6%); 40mg: 32 (6.6%)
Hair color changeAdverse0 (0%)12mg: 12 (2.5%); 40mg: 24 (5.0%)
Falls and injuriesAdverse100 (20.7%)12mg: 124 (25.7%); 40mg: 123 (25.4%)
Suicidal ideationAdverse31 (6.4%)12mg: 33 (6.8%); 40mg: 45 (9.3%)

Subgroup Analysis

Exploratory subgroup analyses did not suggest that the effect of verubecestat on CDR-SB was altered by baseline APOE4 gene-carrier status, age, sex, MMSE score, or PET SUVR. Cognitive worsening appeared by week 13 and did not progress further relative to placebo.


Criticisms

  • Trial terminated early for futility limiting statistical power for some analyses
  • Cognitive worsening may reflect BACE-1 inhibition effects on synaptic function beyond amyloid
  • BACE-2 inhibition may contribute to adverse effects (uncertain physiologic functions)
  • Disease-modifying effect requires longer follow-up than 104 weeks to fully assess
  • Prodromal stage may already be too late in disease course for amyloid-lowering benefit
  • Only 47-49% of patients completed the trial due to early termination
  • Cerebrospinal fluid substudy limited to 5-6 patients per group
  • Higher discontinuation rate in verubecestat groups due to adverse events
  • Amyloid reduction achieved but did not translate to clinical benefit

Funding

Merck Sharp & Dohme

Based on: Verubecestat-Prodromal (New England Journal of Medicine, 2019)

Authors: Michael F. Egan, James Kost, Tiffini Voss, ..., David Michelson

Citation: N Engl J Med 2019;380:1408-20

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