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ADAGIO

A Double-Blind, Delayed-Start Trial of Rasagiline in Parkinson’s Disease

Year of Publication: 2009

Authors: C. Warren Olanow, Olivier Rascol, Robert Hauser, ..., Eduardo Tolosa

Journal: New England Journal of Medicine

Citation: N Engl J Med 2009;361:1268-1278

Link: https://doi.org/10.1056/NEJMoa0809335


Clinical Question

Does early initiation of rasagiline delay disease progression in Parkinson’s disease compared to delayed initiation?

Bottom Line

Rasagiline 1 mg/day met the delayed-start superiority criteria in early PD, suggesting possible disease-modifying effect (UPDRS total: -1.7; P=0.01 at 72 weeks). However, 2 mg/day did NOT meet criteria. Published NEJM 2009. 1176 patients. The inconsistent dose-response weakened the disease-modification interpretation.

Major Points

  • Rasagiline 1mg met all 3 delayed-start criteria: superiority of early vs delayed start at 72 weeks (-1.7; P=0.01).
  • Rasagiline 2mg did NOT meet criteria — inconsistent dose-response undermined disease-modification claim.
  • 1176 early PD patients. Delayed-start design: 36 weeks rasagiline vs placebo → 36 weeks all on rasagiline.
  • Three hierarchical endpoints: (1) superiority at 72 weeks, (2) superiority at 36 weeks, (3) non-inferiority slope test.
  • 1mg met all 3; 2mg met endpoints 2 and 3 but failed endpoint 1.
  • Symptomatic benefit at 36 weeks: -2.82 (1mg) and -3.64 (2mg) vs placebo (both P<0.001).
  • FDA ultimately did not grant disease-modification label due to dose inconsistency.
  • Published NEJM 2009 (Olanow et al.). Teva sponsored.
  • Rasagiline: irreversible MAO-B inhibitor. 1mg/day is the approved PD dose.
  • Highlighted challenges of demonstrating disease modification in PD using delayed-start designs.

Design

Study Type: Randomized, double-blind, placebo-controlled delayed-start trial

Randomization: 1

Blinding: Participants and investigators were blinded

Follow-up Duration: 72 weeks

Centers: 129

Countries: 14 countries (not listed)

Sample Size: 1176

Analysis: Mixed-model repeated-measures ANCOVA; hierarchical testing with Hochberg correction


Inclusion Criteria

  • Age 30–80 years
  • Untreated idiopathic Parkinson’s disease
  • At least two of three cardinal features: tremor, bradykinesia, rigidity
  • Disease duration <18 months
  • Hoehn and Yahr stage <3

Exclusion Criteria

  • Previous antiparkinsonian meds >3 weeks
  • Use of rasagiline/selegiline or coenzyme Q10 within 120 days
  • Atypical parkinsonism
  • Severe psychiatric or cognitive illness

Arms

FieldEarly-start rasagiline 1 mgControlEarly-start rasagiline 2 mgControl
InterventionRasagiline 1 mg/day for 72 weeksPlacebo for 36 weeks, then rasagiline 1 mg/day for 36 weeksRasagiline 2 mg/day for 72 weeksPlacebo for 36 weeks, then rasagiline 2 mg/day for 36 weeks
Duration72 weeks72 weeks72 weeks72 weeks

Outcomes

OutcomeTypeControlInterventionHR / OR / RRP-value
Change in total UPDRS from baseline to 72 weeksPrimary4.50 (delayed-start 1 mg)2.82 (early-start 1 mg)0.02
UPDRS change weeks 12–36 (slope)Secondary0.14 points/week0.09 points/week0.01
UPDRS change in phase 1 (0–36 weeks)Secondary4.27 (placebo)1.26 (rasagiline 1 mg)<0.001
HeadacheAdverse6.2%4.9%
DepressionAdverse6.1%3.5%
FatigueAdverse2.9%5.9%
Back painAdverse5.4%4.9%

Subgroup Analysis

In patients with higher baseline UPDRS (>25.5), both doses met all endpoints. 2 mg showed benefit in this group with 3.63 point lesser worsening (P=0.04).


Criticisms

  • 1 mg showed positive results while 2 mg did not, raising questions about consistency
  • Placebo phase might have been too short to reveal disease modification
  • Primary endpoint (slope analysis) not widely validated
  • Post hoc subgroup analysis supports 2 mg but is exploratory

Funding

Teva Pharmaceutical Industries

Based on: ADAGIO (New England Journal of Medicine, 2009)

Authors: C. Warren Olanow, Olivier Rascol, Robert Hauser, ..., Eduardo Tolosa

Citation: N Engl J Med 2009;361:1268-1278

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