← Back
NeuroTrials.ai
Neurology Clinical Trial Database

ELLDOPA

Effects of Levodopa on the Progression of Parkinson’s Disease

Year of Publication: 2004

Authors: Ira Shoulson, Stanley Fahn, Ken Marek, ..., Karl Kieburtz

Journal: New England Journal of Medicine

Citation: N Engl J Med 2004;351:2498–2508. doi:10.1056/NEJMoa033447

Link: https://doi.org/10.1056/NEJMoa033447


Clinical Question

Does initiating levodopa earlier in the course of Parkinson’s disease modify disease progression or primarily offer symptomatic benefit?

Bottom Line

Levodopa treatment in early PD improves clinical outcomes in a dose-dependent fashion, but imaging showed increased dopamine transporter loss, suggesting no disease-modifying benefit and potential neurotoxicity.

Major Points

  • 361 patients with early PD randomized to placebo or carbidopa–levodopa at 150, 300, or 600 mg/day for 40 weeks
  • After 40 weeks, treatment was stopped and patients were evaluated 2 weeks later to assess for potential disease-modifying effects independent of symptomatic benefit
  • UPDRS scores showed less worsening (or net improvement) in levodopa groups, particularly at 600 mg/day
  • SPECT imaging using [123I]β-CIT revealed greater dopamine transporter loss in the levodopa-treated groups, with a dose-response relationship
  • Dyskinesias were more frequent with higher doses of levodopa (16.5% at 600 mg/day)
  • Trial raised questions about potential neurotoxicity of levodopa despite clear symptomatic benefit

Design

Study Type: Randomized, double-blind, placebo-controlled, dose-ranging trial with imaging substudy

Randomization: 1

Blinding: Participants and investigators were blinded to treatment allocation

Enrollment Period: 1999–2002

Follow-up Duration: 42 weeks (40 weeks treatment + 2-week washout)

Centers: 36

Countries: USA

Sample Size: 361

Analysis: ANCOVA for primary outcome; linear regression for imaging substudy; intention-to-treat


Inclusion Criteria

  • Age ≥30 years
  • Diagnosis of idiopathic Parkinson's disease within 2 years
  • Modified Hoehn–Yahr stage <3
  • Not expected to require symptomatic therapy within 9 months
  • No prior treatment with dopaminergic therapy

Exclusion Criteria

  • Atypical or secondary parkinsonism
  • Prior dopaminergic treatment
  • Significant comorbid medical or psychiatric illness
  • Surgical contraindications
  • Contraindications to SPECT imaging (for substudy participants)

Arms

FieldControlLevodopa 150 mg/dayLevodopa 300 mg/dayLevodopa 600 mg/day
InterventionPlacebo tablets for 40 weeksCarbidopa–levodopa 25/100 mg TID (150 mg/day total)Carbidopa–levodopa 25/100 mg QID (300 mg/day total)Carbidopa–levodopa 25/100 mg QID + one additional tablet (600 mg/day total)
Duration40 weeks treatment + 2-week washout40 weeks treatment + 2-week washout40 weeks treatment + 2-week washout40 weeks treatment + 2-week washout

Outcomes

OutcomeTypeControlInterventionHR / OR / RRP-value
Change in UPDRS total score from baseline to week 42 (2 weeks after stopping treatment)Primary+7.8 points150 mg: +1.9; 300 mg: +0.4; 600 mg: –1.4<0.001 (600 mg vs placebo)
Change in [123I]β-CIT SPECT uptake (caudate + putamen)Secondary−1.4%−7.2% (levodopa groups combined)0.036
Proportion with dyskinesiaSecondary3.3%600 mg: 16.5%
DyskinesiaAdverse3.3%16.5% (600 mg)
NauseaAdverse9%17–20%
Orthostatic symptomsAdverse5%10–12%

Subgroup Analysis

Greater symptomatic benefit seen at higher doses; no evidence of disease modification based on imaging or washout results


Criticisms

  • Two-week washout may not have fully eliminated symptomatic effects of levodopa
  • SPECT imaging changes may reflect pharmacologic downregulation rather than true neurotoxicity
  • No long-term follow-up beyond 42 weeks to assess durability
  • Imaging results apply only to substudy cohort (n=119)

Funding

National Institute of Neurological Disorders and Stroke (NINDS); Department of Veterans Affairs

Based on: ELLDOPA (New England Journal of Medicine, 2004)

Authors: Ira Shoulson, Stanley Fahn, Ken Marek, ..., Karl Kieburtz

Citation: N Engl J Med 2004;351:2498–2508. doi:10.1056/NEJMoa033447

Content summarized and formatted by NeuroTrials.ai.