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PARADIGM

Safety and Efficacy of PrimeC in Amyotrophic Lateral Sclerosis: The PARADIGM Randomized Clinical Trial

Year of Publication: 2026

Authors: Merit Cudkowicz, MD, MSc; Vivian E. Drory, ..., PhD; Jeremy Shefner

Journal: JAMA Neurology

Citation: JAMA Neurol. 2026. doi:10.1001/jamaneurol.2026.0230

Link: https://jamanetwork.com/journals/jamaneu...eurol.2026.0230


Clinical Question

Is PrimeC (celecoxib + ciprofloxacin), a fixed-dose combination targeting neuroinflammation, iron homeostasis, and microRNA dysregulation, safe and well tolerated in people with ALS, and does it show signals of clinical and biomarker efficacy?

Bottom Line

PrimeC was safe and well tolerated over 18 months. Although not powered for efficacy, continuous PrimeC treatment was associated with slower functional decline (ALSFRS-R difference 7.92 points at 18 months, P=.007), reduced ALS complications (HR 0.36, P=.02), and significant modulation of iron-regulatory and microRNA biomarkers, supporting further investigation in a phase 3 trial.

Major Points

  • Phase 2b randomized, double-blind, placebo-controlled trial with 12-month open-label extension
  • PrimeC is a fixed-dose combination of celecoxib (COX-2 inhibitor) and ciprofloxacin (iron chelation + microRNA modulation) in synchronized extended-release formulation
  • 68 participants with definite/probable ALS, randomized 2:1 to PrimeC or placebo
  • Primary outcome was safety — adverse event rates comparable between groups (66.7% vs 65.2%)
  • At month 6 (double-blind), ALSFRS-R difference was 2.23 points (P=.12, not significant)
  • At month 18, participants continuously on PrimeC showed 7.92-point ALSFRS-R advantage over those switched from placebo (P=.007)
  • Bulbar subdomain showed the largest benefit: 3.18-point difference at month 18 (P=.001)
  • ALS complication-free survival significantly favored PrimeC (HR 0.36, 95% CI 0.15-0.85, P=.02)
  • Overall survival trend favoring PrimeC (HR 0.42, P=.11) but not statistically significant
  • Transferrin levels preserved with PrimeC (P=.03); ferritin levels stabilized vs progressive decline in placebo
  • ALS-associated microRNAs (miR-199a-3p/5p, miR-181a-5p, miR-181b-5p) significantly downregulated with PrimeC
  • NfL levels did not differ between groups
  • Primary biomarker outcome (neuron-derived exosomal TDP-43/PgJ2) not yet reported

Design

Study Type: Randomized, double-blind, placebo-controlled, phase 2b trial with open-label extension

Randomization: 1

Blinding: Participants, investigators, site personnel, and sponsor blinded throughout study including open-label extension

Enrollment Period: May 2022 – November 2023

Follow-up Duration: 18 months (6 months double-blind + 12 months open-label extension)

Centers: 4

Countries: Italy, Canada, Israel

Sample Size: 68

Analysis: Mixed models for repeated measures (MMRM) with treatment group, visit, treatment-by-visit interaction, TRICALS risk profile, and background therapy as covariates; intention-to-treat


Inclusion Criteria

  • Adults aged 18–75 years
  • Definite or probable ALS by revised El Escorial criteria
  • Disease duration ≤30 months from symptom onset
  • Slow vital capacity (SVC) ≥60% of predicted value
  • Preenrollment ALSFRS-R slope ≥0.3 points per month decline
  • ALSFRS-R total score ≥25 at screening

Exclusion Criteria

  • Contraindication to PrimeC excipients (celecoxib or ciprofloxacin)
  • Use of feeding tube
  • Invasive ventilator dependence
  • Use of monoamine oxidase inhibitors
  • History of prolonged QT interval or congenital long QT syndrome

Arms

FieldPrimeCControl
InterventionPrimeC tablets (celecoxib 136 mg + ciprofloxacin 1360 mg modified release) administered twice daily, in addition to standard-of-care ALS treatmentsMatching placebo tablets twice daily for 6 months, then switched to PrimeC for 12-month open-label extension
Duration18 months (6 months double-blind + 12 months open-label extension)6 months placebo, then 12 months PrimeC

Outcomes

OutcomeTypeControlInterventionHR / OR / RRP-value
Safety and tolerability: incidence and severity of treatment-emergent adverse eventsPrimary65.2%66.7%Not significant (comparable rates)
SecondaryNot separately reportedNot separately reported.12
SecondaryNot separately reportedNot separately reported.007
SecondaryNot separately reportedNot separately reported.001
SecondaryReferenceHR 0.36.02
SecondaryReferenceHR 0.42.11
Any AEAdverse65.2%66.7%
Drug-related TEAEAdverse4.3%20.0%
Serious TEAEAdverse13.0%8.9%
TEAE Leading to DeathAdverse8.7%4.4%
GI DisordersAdverse4.3%24.4%
InfectionsAdverse17.4%13.3%

Criticisms

  • Phase 2b trial not powered for clinical efficacy — all efficacy results are exploratory
  • Small sample size (n=68) limits statistical power and generalizability
  • No multiplicity adjustment for secondary and exploratory endpoints
  • Primary biomarker outcome (exosomal TDP-43/PgJ2) not yet reported
  • Open-label extension design means all participants received PrimeC after 6 months — convergence of trajectories is expected with delayed-start paradigms
  • ALSFRS-R slope requirement (≥0.3 pts/month) and disease duration ≤30 months may exclude slow progressors
  • Imbalanced randomization (2:1) reduces power of between-group comparisons
  • Sponsored and designed by drug manufacturer (NeuroSense Therapeutics)

Funding

NeuroSense Therapeutics

Based on: PARADIGM (JAMA Neurology, 2026)

Authors: Merit Cudkowicz, MD, MSc; Vivian E. Drory, ..., PhD; Jeremy Shefner

Citation: JAMA Neurol. 2026. doi:10.1001/jamaneurol.2026.0230

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