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N-MOmentum

Inebilizumab for the treatment of neuromyelitis optica spectrum disorder (N-MOmentum): a double-blind, randomised placebo-controlled phase 2/3 trial

Year of Publication: 2019

Authors: Bruce A C Cree, Jeffrey L Bennett, Ho Jin Kim, ..., Eliezer Katz

Journal: The Lancet

Citation: Lancet 2019; 394: 1352-1363

Link: https://doi.org/10.1016/S0140-6736(19)31817-3


Clinical Question

Does inebilizumab, an anti-CD19 B cell-depleting antibody, reduce the risk of NMOSD attacks and disability compared to placebo in patients with neuromyelitis optica spectrum disorder?

Bottom Line

Inebilizumab significantly reduced the risk of NMOSD attacks by 73% compared to placebo (HR 0.272), with benefits also seen in disability worsening, MRI lesion activity, and disease-related hospitalizations. The trial was stopped early due to clear efficacy.

Major Points

  • First large randomized controlled trial demonstrating superiority of a B cell-depleting therapy over placebo in NMOSD
  • 73% relative risk reduction in NMOSD attacks with inebilizumab (12% vs 39%, p<0.0001)
  • Number needed to treat of 3.73 to prevent one attack over 197 days
  • Trial stopped early by independent data-monitoring committee due to clear efficacy (conditional power >99%)
  • 93% of participants were AQP4-IgG seropositive; similar efficacy in this subgroup (HR 0.227)
  • Inebilizumab depleted CD20 B cells to <10% of baseline within 4 weeks
  • No significant difference in low-contrast visual acuity despite reduced optic neuritis attacks
  • Safety profile similar to placebo during randomized controlled period; two deaths occurred in open-label extension

Design

Study Type: Phase 2/3 double-blind randomized placebo-controlled trial with open-label extension

Randomization: 1

Blinding: Double-blind; participants, investigators, sponsor, adjudication committee, and all clinical staff including EDSS assessors were masked; inebilizumab and placebo were indistinguishable in appearance

Enrollment Period: January 6, 2015 to September 24, 2018

Follow-up Duration: Up to 197 days (randomized controlled period)

Centers: 99

Countries: Australia, Bulgaria, Canada, Colombia, Czech Republic, Estonia, Germany, Hong Kong, Hungary, Israel, Japan, Mexico, Moldova, New Zealand, Peru, Poland, Russia, Serbia, South Africa, South Korea, Spain, Taiwan, Thailand, Turkey, USA

Sample Size: 230

Analysis: Intention-to-treat; Cox proportional hazards regression for primary endpoint; logistic regression for EDSS worsening; ANCOVA for visual acuity; negative binomial regression for MRI lesions and hospitalizations; Bonferroni-based chain procedure for type 1 error control; SAS version 9.3


Inclusion Criteria

  • Adults ≥18 years old
  • Diagnosis of NMOSD (AQP4-IgG seropositive or seronegative meeting Wingerchuk 2006 criteria)
  • Expanded Disability Status Scale (EDSS) score ≤8.0
  • History of ≥1 attack requiring rescue therapy (IV corticosteroids, IVIG, plasma exchange) in year before screening, OR ≥2 attacks requiring rescue therapy in 2 years before screening
  • Written informed consent

Exclusion Criteria

  • Abnormal baseline laboratory values
  • Positive tuberculosis test
  • Hepatitis
  • Contraindicated health condition
  • Not stable after recent attack
  • Receipt of contraindicated medications in previous 6 months
  • Unable or unwilling to participate
  • Unable to undergo MRI
  • EDSS score >8.0

Arms

FieldInebilizumabControl
InterventionInebilizumab 300mg IV on day 1 and day 15 (total dose 600mg); all participants received oral corticosteroids (prednisone 20mg/day or equivalent) days 1-14, tapered to day 21Placebo IV on day 1 and day 15; all participants received oral corticosteroids (prednisone 20mg/day or equivalent) days 1-14, tapered to day 21
DurationUp to 197 days or until adjudicated attackUp to 197 days or until adjudicated attack

Outcomes

OutcomeTypeControlInterventionHR / OR / RRP-value
Time (in days) from day 1 to onset of an adjudicated NMOSD attack on or before day 197Primary22/56 (39%) had attack21/174 (12%) had attack0.272<0.0001
EDSS worsening from baseline at last visitSecondary19/56 (34%)27/174 (16%)0.370.0049
Change in low-contrast visual acuity binocular score from baselineSecondaryLSM 1.442 (SE 1.217)LSM 1.576 (SE 0.935)0.90
Cumulative number of active MRI lesions from baselineSecondaryMean 2.3 (SD 1.3), n=32Mean 1.6 (SD 1.0), n=79RR 0.5660.0034
Cumulative number of NMOSD-related inpatient hospitalizationsSecondaryMean 1.4 (SD 0.7), n=8Mean 1.0 (SD 0.0), n=10RR 0.2860.010
Primary endpoint in AQP4-IgG seropositive subgroupSecondary22/52 (42%) had attack18/161 (11%) had attack0.227<0.0001
Any adverse eventAdverse41/56 (73%)125/174 (72%)
Any serious adverse eventAdverse5/56 (9%)8/174 (5%)
Urinary tract infectionAdverse5/56 (9%)20/174 (11%)
ArthralgiaAdverse2/56 (4%)17/174 (10%)
Infusion-related reactionAdverse6/56 (11%)16/174 (9%)
Back painAdverse2/56 (4%)13/174 (7%)
HeadacheAdverse4/56 (7%)13/174 (7%)
NasopharyngitisAdverse6/56 (11%)13/174 (7%)
DepressionAdverse5/56 (9%)4/174 (2%)
Grade 3 neutropeniaAdverse0/56 (0%)3/174 (2%)
Deaths during RCPAdverse00
Deaths during open-label extensionAdverse1 (originally placebo)1 (originally inebilizumab)

Subgroup Analysis

Treatment effect was consistent across optic nerve and spinal cord domains; consistent across unanimous vs majority adjudication committee decisions; consistent across investigator-suspected attacks; consistent across racial/ethnic subgroups (white, non-white, Asian, non-Asian) and BMI categories. Efficacy could not be determined in AQP4-IgG seronegative patients due to small numbers (n=17, only 4 in placebo group with no attacks).


Criticisms

  • Did not meet secondary endpoint for low-contrast visual acuity despite reducing optic neuritis attacks, suggesting inebilizumab may not reduce attack severity or facilitate recovery
  • Floor effect in placebo group (27% read 0/70 characters on low-contrast chart at baseline) limited ability to detect visual acuity changes
  • Only 17 AQP4-IgG seronegative patients enrolled (7%), with only 4 in placebo group - efficacy in seronegative NMOSD could not be determined
  • Strict exclusion criteria (comorbidities, lab abnormalities) may limit generalizability to broader NMOSD population
  • Relatively small study (230 participants) with short randomized controlled period (6.5 months)
  • Study stopped early before reaching target enrollment (252) and target attacks (67)
  • One death in open-label phase could not definitively exclude treatment relationship; differential included ADEM, atypical NMOSD attack, and PML
  • Long-term safety data beyond 6.5 months not available from randomized controlled period
  • 3:1 randomization resulted in small placebo group (n=56) with limited precision

Funding

MedImmune and Viela Bio

Based on: N-MOmentum (The Lancet, 2019)

Authors: Bruce A C Cree, Jeffrey L Bennett, Ho Jin Kim, ..., Eliezer Katz

Citation: Lancet 2019; 394: 1352-1363

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